Elhízás: ki dolgozik rajta

Intézményenként egy kártya. Célpontra vagy országra szűkítheted a listát.

• Minden célpont
• GLP-1 172
• PPAR 0
• FTO 0
• AMPK 3
• GIP 20
• SGLT2 5
• STAT3 0
• SIRT1 0
• PI3K 0
• NRF2 0
• AKT 0
• mTOR 2
• NLRP3 3
• MAPK 0
• TP53 0
• TNF 0
• IL-6 1
• TLR4 0
• BDNF 2
• DPP-4 1
• JNK 0
• APOE 0
• ERK 0
• IL-17 1
• PTEN 0
• STING 0
• ACE2 0
• HIF-1 0
• PCSK9 0
• VEGF 0
• IL-1 0
• JAK 0
• PNPLA3 2
• YAP 0
• HMGB1 0
• CTNNB1 0
• HER2 0
• MMP-9 0
• PD-L1 0
• PINK1 0
• TFEB 0
• TREM2 0
• CCR5 0
• FGFR 0
• GPX4 0
• IGF1R 0
• IL-23 0
• METTL3 0
• PD-1 0
• STK11 0
• AR 0
• BCL-2 0
• CFTR 0
• COX-2 0
• HDAC 0
• EGFR 0
• HSP90 0
• MEK 0
• MYC 0
• PKM2 0

Funded research projects that name this disease in their title, terms or summary and have not ended yet: who is funded, where, how much and until when. Organisations only; the funder's page has the details.

• NIH NIAID · F30

  The role of Aiolos in lung-resident CD8+ memory T cell programming.

  Ohio State University· Columbus, Egyesült Államok· 45 821 USD awarded in fiscal year 2026· until 2030. okt.

• NIH NICHD · F32

  Role of the Placental Intrinsic Clock as a Central Regulator of Endocrine Function and Offspring Growth

  University of Minnesota· Minneapolis, Egyesült Államok· 76 300 USD awarded in fiscal year 2026· until 2029. jún.

• NIH NIAID · F31

  Dysregulation of Aging Regulatory T-Cell Response in Autoimmune Disease

  Utah State Higher Education System--University of Utah· Salt Lake City, Egyesült Államok· 38 914 USD awarded in fiscal year 2026· until 2029. okt.

• NIH NIA · R01

  The effects of sleep and diet on glial function

  Texas A&m University· College Station, Egyesült Államok· 527 686 USD awarded in fiscal year 2026· until 2031. máj.

• NIH NIDDK · RC2

  Interorgan Genomic Atlas for Discovery of Bioenergetic Dynamics and Mechanisms of Time-Restricted Feeding

  Northwestern University· Chicago, Egyesült Államok· 1 641 136 USD awarded in fiscal year 2026· until 2031. jún.

• European Commission (Horizon Europe) · HORIZON-TMA-MSCA-PF-EF

  Elucidating the role of maternal nutrient stores in the metabolism of the preimplantation embryo (MaterMeta)

  Kobenhavns Universitet· Kobenhavn, Dánia· EU contribution 247 553 EUR· until 2029. aug.

  The disease is named only in the funder's project summary, not in the title.

• European Commission (Horizon Europe) · HORIZON-TMA-MSCA-PF-EF

  Studying the role of Tbx-3 expressing neurons during obesity and ageing (OBrAgeing)

  Institut National de la Sante Et de la Recherche Medicale· Paris, Franciaország· EU contribution 226 421 EUR· until 2029. aug.

• European Commission (Horizon Europe) · HORIZON-ERC

  Selective Sensory Vagal Interface: a bionic platform for internal organ modulation (VisceraLink)

  Medizinische Universitaet Wien· Wien, Ausztria· EU contribution 2 210 625 EUR· until 2031. dec.

  The disease is named only in the funder's project summary, not in the title.

• European Commission (Horizon Europe) · HORIZON-TMA-MSCA-DN

  Metabolic disease heterogeneity definition, prevention and remission with targeted interventions (MEDIculous REMeDy)

  Helmholtz Zentrum Muenchen Deutsches Forschungszentrum Fuer Gesundheit und Umwelt Gmbh· Neuherberg, Németország· EU contribution 4 437 236 EUR· until 2030. dec.· 11 countries

  The disease is named only in the funder's project summary, not in the title.

• European Commission (Horizon Europe) · HORIZON-TMA-MSCA-DN

  Bridging Chronobiology and Physiology through Expert Training (ChronoBridge)

  Ecole Polytechnique Federale de Lausanne· Lausanne, Svájc· EU contribution 3 885 022 EUR· until 2030. dec.· 9 countries

  The disease is named only in the funder's project summary, not in the title.

10 of 4093 running projects, newest first. Listed amounts together: 2,5 Mrd USD + 430,1 M EUR.

Sources: NIH RePORTER (U.S. National Institutes of Health, public domain) and CORDIS (European Commission, CC BY 4.0). Changes: filtered by disease, organisations matched to ROR.

Vizsgálatok, amelyek regiszteradata szerint az egyéni résztvevői adatokat (IPD) megosztják.

Ebben a nézetben egy vizsgálat sem jelzi, hogy megosztaná a résztvevői adatokat.

Publikációk erről a betegségről egy MeSH-módszerkifejezés szerint, például szekvenálási vagy képalkotó módszerre.

• Single-Cell Analysis 21
• Organoids 10
• Mendelian Randomization Analysis 180
• CRISPR-Cas Systems 7
• Gene Expression Profiling 144
• Sequence Analysis, RNA 10
• Whole Genome Sequencing 4
• Exome Sequencing 19
• Proteomics 127
• Metabolomics 191
• Mass Spectrometry 28
• Flow Cytometry 16
• Immunohistochemistry 41
• Xenograft Model Antitumor Assays 15
• Positron-Emission Tomography 26
• Magnetic Resonance Imaging 258
• Machine Learning 155
• Deep Learning 20
• Cohort Studies 1014
• Genome-Wide Association Study 203

• Bariatric surgery and subsequent weight loss lead to a reversal of peripheral blood monocytosis in obesity.

  Int J Obes (Lond), 2026. ápr.

• γδ T cell exhaustion is associated with obesity in individuals with type 2 diabetes.

  Int J Obes (Lond), 2026. ápr.

• Circulating CD16+ CD56+ Natural Killer Cells in Colorectal Cancer and Their Correlation with Survival.

  Malawi Med J, 2026. jan.

• Probiotic Enterococcus faecalis AG5 mitigated high fat diet induced obesity and produced propionic acid stimulated apoptosis in 3T3-L1 pre-adipocyte.

  Life Sci, 2020. aug.

• Metformin improves salivary gland inflammation and hypofunction in murine Sjögren's syndrome.

  Arthritis Res Ther, 2019. jún.

• γδ T cells are required for pulmonary IL-17A expression after ozone exposure in mice: role of TNFα.

  PLoS One, 2014. máj.

• Metformin, a diabetes drug, eliminates tumor-initiating hepatocellular carcinoma cells.

  PLoS One, 2013. júl.

• An increase in the Akkermansia spp. population induced by metformin treatment improves glucose homeostasis in diet-induced obese mice.

  Gut, 2013. jún.

• Exercise and caloric restriction alter the immune system of mice submitted to a high-fat diet.

  Mediators Inflamm, 2013. márc.

• Oral or parenteral administration of curcumin does not prevent the growth of high-risk t(4;11) acute lymphoblastic leukemia cells engrafted into a NOD/SCID mouse model.

  Int J Oncol, 2012. dec.

Megmutatjuk, a betegség mely célpontjait említi az absztrakt, és onnan a rajtuk dolgozókhoz jutsz.